Synkinesis
Evaluation & Treatment of Synkinesis
Synkinesis means “simultaneous movement.”
Synkinesis occurs secondary to abnormal facial nerve
regeneration after Bell’s palsy or instances where
the facial nerve has been cut and sewn back together.
The facial nerve fibers can implant into the different
muscles in cases of bells palsy. Additionally, when
the nerve is re-sewn, the facial nerve fibers oftentimes
reconnect to the wrong nerve group causing undesired
and simultaneous facial movement. Synkinesis, therefore,
results in abnormal synchronization of facial movement
where muscles, other than those intended contract together
during a particular movement pattern.
Synkinesis does have some predictable facial muscle
patterns and can have a range of severities. It is important
to separate true paralysis and synkinesis. If patients
have good facial tone and some visible movement, then
they do not have full paralysis and many of their abnormal
facial movements are a result of synkinesis. The most
common effect of synkinesis is when patients experience
eye closure during a smile. The eyes tend to twitch
or close while the patient is trying to smile or laugh.
Synkinesis can also be a powerful cause of inability
of the corner of the mouth to move upwards in patients
who have regained their facial tone. Patients usually
just think that their face is not moving; however, in
many patients their inability to smile is secondary
to synkinetic (simultaneous) movement of muscles that
droop the corner of mouth (depressor anguli oris, platysma,
and mentalis muscles) and muscles that elevate the area
(zygomaticus major and minor). Other patterns of synkinesis
are dimpling in the chin and narrowing of the eyes.
In addition to these abnormal movement patterns, synkinesis
also causes increased muscle tone with spasm, contracture
and tightness of the neck bands and cheeks.
PREVENTION
There are some ways to reduce the risk of developing
synkinesis after bells palsy. The Chevalier’s
method is one of the more common ways of using a “facial
re-education” method to prevent synkinesis and
educate your facial muscles. Patients are encouraged
to maintain facial symmetry by keeping the normal side
of face up when speaking, chew food with eyes open,
avoid gum, wear sunglasses to prevent squinting, massage
the intraoral buccal area, always align face to block
associated movement, Stretch orbicularis oculi (eye
muscles). Patients are also encouraged to really work
on having slow and symmetric movements. The key aspect
of prevention is the first 3-4 months after injury or
Bell’s palsy. It is important to note that some
studies have shown that electrical stimulation can result
in increased likelihood of developing synkinesis.
TREATMENT
Once synkinesis has occurred, treatment relies on three
distinct modalities: neuromuscular retraining (physical
therapy), Botox (botulinum toxin) and surgery. Treatment
of synkinesis can be initiated at any time after its
occurrence. This may be even years after a patient has
suffered Bell’s palsy or facial paralysis.
Neuromuscular retraining and physical therapy of synkinesis
is very different than what is performed for other medical
problems such as back pain and orthopedic injuries.
Facial neuromuscular retraining is more comparable to
a vocal therapist that is treating a singer who has
hoarseness or poor mechanics. Facial neuromuscular retraining
is primarily focused on coordinating appropriate facial
muscle movements. This is achieved by inhibiting the
activity of the abnormal movement patterns resulting
in “auto-paralysis” of unwanted muscles.
The muscles that are contracting abnormally are first
identified. Muscles that are contracting out of sequence
are inhibited. Small steps are usually taken in order
to gradually retrain the muscles as there needs to be
significant changes at the neurologic (brain) level
for success. Electrical stimulation is avoided as it
tends to increase the overactive muscles. Muscles that
are extremely overactive in the cheek and neck are actively
massaged and stretched. Patients are discouraged from
undergoing strong muscle strengthening exercises as
again this is more about re-coordination rather than
stimulation. Patients are also taught how to elevate
the upper eyelids during eating to reduce the eye synkinesis.
Ninety percent of the therapy is done with the patient
at home. The therapist at the Facial Paralysis Institute
typically teaches the patients the appropriate home
exercises. Other treatment modalities will focus on
mirror and video exercises.
The second mode of therapy for synkinesis is BOTOX
(botulinum toxin-A). Botox is used in conjunction with
facial neuromuscular therapy in most cases. Botox works
by reducing the activity of the muscles that are overactive
or uncoordinated. Most common areas of injection are
eye muscles (orbicularis), neck bands (platysma), and
chin dimpling (mentalis). We also utilize it occasionally
for very tight cheek if therapy has been unsuccessful.
Botox can also be used to symmetrize the face by reducing
the activity of certain muscles on the normal side of
the face such as: forehead, lower lip depressors (depressor
anguli oris) and crow’s feet (orbicularis).
The final modality for synkinesis is surgery. Surgery
is utilized only when physical therapy and botox have
been unsuccessful in obtaining the desired results.
Static suspension of the corners of the mouth, surgical
manipulation of the neck bands and blepharoplasty (eyelid
surgery) are commonly utilized to address these concerns.
During your consultation, all options are discussed
at length if you are an appropriate candidate. |